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Thursday, April 26, 2012
Facebook for Windows Phone getting a major refresh
The Facebook app for Windows Phone is certainly serviceable, but it's never really been anything to get excited about. In fact, it wasn't really something you looked forward to using and it's been slow to add the features considered standard on competing mobile platforms. Well, with the soon to land version 2.5, that capability gap will get quite a bit smaller. It hasn't hit the Marketplace yet, but when it does it'll bring support for group chats, threaded messaging, tagging in posts and the ability to like or delete not only posts but comments as well. The photo pages have also gotten a visual refresh and you can now tap on links in posts to go straight to the relevant content. For a few more details hit up the source, and keep checking the Marketplace for updates -- Facebook promises it won't be long now.
Facebook for Windows Phone getting a major refresh originally appeared on Engadget on Tue, 24 Apr 2012 09:56:00 EDT. Please see our terms for use of feeds.
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Wednesday, April 25, 2012
Google gets back into the phone sales business, offering Galaxy Nexus for $399
The rumors were true -- Google once again is selling unlocked smartphones on its own, with the Samsung Galaxy Nexus going for $399 in the Google Play Store.
The phone Google's offering is an unlocked pentaband GSM device, same as what many of us had imported previously. The "pentaband" part means that it'll work on AT&T and T-Mobile 3G/4G networks. And because it's unlocked, you can take it out of the country and pop in another carrier's SIM card without needing an unlock code.
Google's tried selling phones before, starting with the original Nexus One in early 2010. That ended poorly, with customer support issues dogging Google, HTC and T-Mobile (which at launch was the only carrier to officially support the N1). Google finally shut down the phone store in May 2010 and said the Nexus One would come to retail stores. That didn't happen. Later, the remaining Nexus One stock was offered as a developer device.
So what happened? Google's Mario Queiroz told us at Google IO in May 2010 that "The web store as a direct distribution model is really a very small part of the overall Android strategy. With the Nexus One we had the objective of bringing to market a very technologically sophisticated phone, which we did, which really set a new bar for quality. And I think you've seen a lot of the phones in the market today, that have come to market in the past few months, have been influencing, in many ways ... by what the Nexus One was able to do."
Sweetening the deal is that Google Wallet -- which for some time has been official only on the Sprint Nexus S, and now on the Sprint Galaxy Nexus -- is preloaded on this GSM device.
For our part, this is the way Google should sell Nexus devices. Unlocked GSM versions, able to work on networks worldwide. And, of course, it's what some are calling the one "true" Nexus because you're able to build for it directly from the Android Open Source Project, with no pesky CDMA licensing restrictions keeping that from happening. (We don't quite subscribe to that "True Nexus" theory ourselves, though.)
Devices in Google Play right now is open only in the U.S. But Andy Rubin, in a blog post this morning, says it'll be coming to more countries. The obvious follow-up question is this, though: Google won't be stopping at selling phones at Google Play, will it?
Buy now: Galaxy Nexus at Google Play
More: Google Mobile Blog
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Tuesday, April 24, 2012
Gut organisms could be clue in controlling obesity risk
ScienceDaily (Apr. 23, 2012) ? The international obesity epidemic is widespread, nondiscriminatory, and deadly. But do we really understand all of the factors underlying this alarming trend? The concept of energy balance (energy consumed = energy expended + energy stored) is undeniable, being driven by the first law of thermodynamics. Consequently, there is no contradiction that excessive calorie intake and plummeting levels of physical activity are largely to blame for our ever-expanding waistlines.
However, scientists remain baffled as to why some individuals are particularly prone to becoming obese and if there is anything aside from lowering calorie consumption and increasing activity levels that can be done to prevent and/or reverse excessive weight gain in our most at-risk populations.
Physiologists have long known that our intestines are brimming with live bacteria, some of which provide important substances (e.g., vitamin B-12) to their host. However, research conducted over the last decade suggests that these organisms, often referred to as intestinal microbiota, may play a far greater role in human health than previously imagined. One area of intense interest is the possibility that the mix of intestinal bacteria with which we are endowed might directly influence our risk for obesity. Obese individuals tend to have different microbial profiles in their intestines than lean individuals, and scientists have learned that the bacteria common to obesity may metabolize the food we eat in a way that allows us to harvest more calories from it and deposit those calories as fat.
To determine whether altering one's bacterial profile can change obesity risk, researchers from the French Institute for Agricultural Research (INRA) transferred the intestinal bacteria of obesity-prone or obesity-resistant rats into the intestinal tracts of germ-free mice recipients, therefore having no innate gut microbiota. Some animals were fed a regular diet, whereas others were provided unlimited access to a high-fat diet. Food intake and weight gain were monitored for 8 weeks, and intestinal samples were analyzed for a variety of physiologic markers of metabolism and normal feedback mechanisms known to play a role in maintenance of energy balance.
As hypothesized, mice that received intestinal bacteria from obesity-prone animals ate more food, gained more weight, and became more obese than those receiving microbiota from obesity-resistant animals. Animals with microbiota transferred from obesity-prone animals also exhibited changes in intestinal nutrient sensors and gut peptide levels, likely influencing how the animals responded to eating.
The authors' conclusions are three-fold. First, they theorize that obese individuals, when given the opportunity to overeat, may harbor specific gut microbiota profiles that promote excess weight gain. Second, they propose that differences in gut microbes can be related to behavioral changes and increased food intake. Finally, they believe that the mix of microbiota you have may influence your ability to properly sense and respond to a meal. They hope to eventually find ways to manipulate the intestinal microbiota profiles of especially at-risk individuals so that they can more easily maintain a healthy body weight.
As part of the scientific program of the American Society for Nutrition, the nation's leading nutrition research society, results from this study were presented on April 23, 2012 in San Diego, CA.
Frank Duca, Yassine Sakar and Mihai Covasa from INRA, France were co-authors on this paper.
This study was funded by the French National Institute for Agricultural Research (INRA).
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The above story is reprinted from materials provided by Federation of American Societies for Experimental Biology (FASEB), via Newswise.
Note: Materials may be edited for content and length. For further information, please contact the source cited above.
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